You Searched For: ADA

Catalog Number: (ABNOH00000103-D01)

Supplier: Abnova

Description: Rabbit polyclonal antibody raised against a full-length human ADAR protein.

UOM: 1 * 100 µl

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Catalog Number: (ANTIA10807-100)

Supplier: ANTIBODIES.COM

Description: Rabbit polyclonal antibody to ADX for WB with samples derived from human.

UOM: 1 * 100 µl

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Catalog Number: (ABNOH00000132-A01)

Supplier: Abnova

Description: Anti-ADK Mouse Polyclonal Antibody

UOM: 1 * 50 µl

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Supplier: Abcam

Description: Rabbit monoclonal [EPR6581] to ADO - BSA and Azide free.

Catalog Number: (USBI031720-AP)

Supplier: US Biological

Description: Anti-AIDA Rabbit Polyclonal Antibody (AP (Alkaline Phosphatase))

UOM: 1 * 200 µl

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Supplier: Abcam

Description: Rabbit monoclonal [EPR4629] to ADX - BSA and Azide free.

Catalog Number: (USBI031653-BIOTIN)

Supplier: US Biological

Description: Anti-ADPRH Rabbit Polyclonal Antibody (Biotin)

UOM: 1 * 200 µl

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Catalog Number: (BOSSBS-2168R-A350)

Supplier: Bioss

Description: ADAR1 converts adenosine to inosine in dsRNA, which destabilizes the dsRNA helix. This activity is important for various functions like site-specific RNA editing of transcripts of the glutamate receptors and modifying viral RNA genomes (which may be responsible for hypermutation of certain negative-stranded viruses, e.g., measles virus). ADAR1 also binds to short interfering RNAs (siRNA) without editing them and suppresses siRNA-mediated RNA interference. This protein is ubiquitously expressed, with the highest levels being found in brain and lung.

UOM: 1 * 100 µl

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Catalog Number: (BOSSBS-0007R-A750)

Supplier: Bioss

Description: AM and PAMP are potent hypotensive and vasodilatator agents. Numerous actions have been reported most related to the physiologic control of fluid and electrolyte homeostasis. In the kidney, am is diuretic and natriuretic, and both am and pamp inhibit aldosterone secretion by direct adrenal actions. In pituitary gland, both peptides at physiologically relevant doses inhibit basal ACTH secretion. Both peptides appear to act in brain and pituitary gland to facilitate the loss of plasma volume, actions which complement their hypotensive effects in blood vessels.

UOM: 1 * 100 µl

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Catalog Number: (BOSSBS-0007R-A488)

Supplier: Bioss

Description: AM and PAMP are potent hypotensive and vasodilatator agents. Numerous actions have been reported most related to the physiologic control of fluid and electrolyte homeostasis. In the kidney, am is diuretic and natriuretic, and both am and pamp inhibit aldosterone secretion by direct adrenal actions. In pituitary gland, both peptides at physiologically relevant doses inhibit basal ACTH secretion. Both peptides appear to act in brain and pituitary gland to facilitate the loss of plasma volume, actions which complement their hypotensive effects in blood vessels.

UOM: 1 * 100 µl

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Catalog Number: (BOSSBS-0007R-A350)

Supplier: Bioss

Description: AM and PAMP are potent hypotensive and vasodilatator agents. Numerous actions have been reported most related to the physiologic control of fluid and electrolyte homeostasis. In the kidney, am is diuretic and natriuretic, and both am and pamp inhibit aldosterone secretion by direct adrenal actions. In pituitary gland, both peptides at physiologically relevant doses inhibit basal ACTH secretion. Both peptides appear to act in brain and pituitary gland to facilitate the loss of plasma volume, actions which complement their hypotensive effects in blood vessels.

UOM: 1 * 100 µl

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Catalog Number: (BOSSBS-2167R-A555)

Supplier: Bioss

Description: Catalyzes the hydrolytic deamination of adenosine to inosine in double-stranded RNA (dsRNA) referred to as A-to-I RNA editing. This may affect gene expression and function in a number of ways that include mRNA translation by changing codons and hence the amino acid sequence of proteins; pre-mRNA splicing by altering splice site recognition sequences; RNA stability by changing sequences involved in nuclease recognition; genetic stability in the case of RNA virus genomes by changing sequences during viral RNA replication; and RNA structure-dependent activities such as microRNA production or targeting or protein-RNA interactions. Can edit both viral and cellular RNAs and can edit RNAs at multiple sites (hyper-editing) or at specific sites (site-specific editing). Its cellular RNA substrates include: bladder cancer-associated protein (BLCAP), neurotransmitter receptors for glutamate (GRIA2) and serotonin (HTR2C) and GABA receptor (GABRA3). Site-specific RNA editing of transcripts encoding these proteins results in amino acid substitutions which consequently alters their functional activities. Exhibits low-level editing at the GRIA2 Q/R site, but edits efficiently at the R/G site and HOTSPOT1. Its viral RNA substrates include: hepatitis C virus (HCV), vesicular stomatitis virus (VSV), measles virus (MV), hepatitis delta virus (HDV), and human immunodeficiency virus type 1 (HIV-1). Exhibits either a proviral (HDV, MV, VSV and HIV-1) or an antiviral effect (HCV) and this can be editing-dependent (HDV and HCV), editing-independent (VSV and MV) or both (HIV-1). Impairs HCV replication via RNA editing at multiple sites. Enhances the replication of MV, VSV and HIV-1 through an editing-independent mechanism via suppression of EIF2AK2/PKR activation and function. Stimulates both the release and infectivity of HIV-1 viral particles by an editing-dependent mechanism where it associates with viral RNAs and edits adenosines in the 5'UTR and the Rev and Tat coding sequence.

UOM: 1 * 100 µl

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Catalog Number: (BOSSBS-11807R-A488)

Supplier: Bioss

Description: Aladin (Adracalin) belongs to a family of WD repeat-containing proteins. These proteins have a wide variety of functions, including signal transduction regulation, RNA processing and transcription. Aladin plays a role in peripheral and central nervous system development. It is widely expressed, with the highest expression seen in pituitary gland, corpus callosum, cerebellum, adrenal gland and gastrointestinal structures. Defects in Aladin cause the autosomal recessive disorder achalasia-addisonianism-alacrima (triple A) syndrome. Triple A syndrome is characterized by achalasia, alacrima and adrenocortico-tropin-resistant adrenal insufficiency. Robust expression in neural systems associated with cognitive, motor and sensory functions is consistent with the myriad of symptoms experienced by patients with triple A syndrome.

UOM: 1 * 100 µl

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Catalog Number: (USBI031644-AP)

Supplier: US Biological

Description: Anti-ADM Rabbit Polyclonal Antibody (AP (Alkaline Phosphatase))

UOM: 1 * 200 µl

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Catalog Number: (USBI031648-HRP)

Supplier: US Biological

Description: Anti-ADO Rabbit Polyclonal Antibody (HRP (Horseradish Peroxidase))

UOM: 1 * 200 µl

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Catalog Number: (ABCAAB1017-250)

Supplier: Abcam

Description: Anti-Aflatoxin B1 Mouse Monoclonal Antibody [clone: AFA-1]

UOM: 1 * 250 µG

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